End-tidal Carbon Dioxide as a Surrogate of Cardiac Output

November 10

by Jonathan Downham

End-tidal Carbon Dioxide as a Surrogate of Cardiac Output

The study entitled “End-tidal carbon dioxide (EtCO₂) is better than arterial pressure for predicting volume responsiveness by the passive leg raising test” published in Intensive Care Medicine has garnered much attention from the field in recent months (see article reviewfrom ESICM NEXT). This attention is due to its probable impact on clinical practice: This study reported that changes in EtCO₂ induced by a passive leg raising (PLR) test predicted fluid responsiveness with a good reliability, while the changes in arterial pulse pressure during PLR did not.

Why do we use vasopressin?

Vasopressin, also known as antidiuretic hormone (ADH), plays a crucial role in regulating blood pressure in critically ill patients through its effects on the cardiovascular system. Here’s how vasopressin helps maintain blood pressure in physiological terms:Vasoconstriction: Vasopressin acts directly on blood vessels, causing them to constrict. This vasoconstrictive effect increases peripheral vascular resistance, which is

CCP Podcast: So you want to be an ACCP?

This is a chat with Rowan Grieves, who works in Belfast, about her journey to become an ACCP and the hurdles she had to overcome when starting this new role.

Cardiac Output

Cardiac output refers to the volume of blood pumped by the heart per minute. It is a measure of the heart’s efficiency in delivering oxygen and nutrients to the body’s tissues. Cardiac output is determined by two factors: heart rate and stroke volume. Heart rate is the number of times the heart beats per minute, while

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End-tidal Carbon Dioxide as a Surrogate of Cardiac Output